Adenosine mediates the amelioration of social novelty deficits during rhythmic light treatment of 16p11.2 deletion female mice.

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Researchers found that flashing lights at 40 Hz (a specific rhythm) helped improve social behavior in female mice with the 16p11.2 deletion by triggering the release of a brain chemical called adenosine, which calmed overactive brain signals in the prefrontal cortex — suggesting this non-invasive light therapy may one day be a treatment option for social difficulties in people with 16p11.2 deletion.